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Stem Cell Therapy for DMD

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Stem Cell Therapy for dmd in India About DMD Duchenne muscular dystrophy, also known as Duchenne muscular dystrophy or simply Duchenne, is a rare genetic disorder. It often affects men, but it can also affect women in rare cases. Duchenne triggers the body's muscles to weaken and deteriorate over time, ultimately leading to death. A variation in the DMD gene, which causes Duchenne, occurs before birth and can be inherited, or new mutations in the gene can occur spontaneously.   Can stem cell therapy cure muscular dystrophy? Stem cells are known to have measurable applications in medicine for Muscular Dystrophy (DMD). By triggering activation, these cells may rebuild affected muscles and provide adequate pain relief. Although the intended worsening of MD patients is around 20% per year, the greatest practising doctors consider the absence of such deterioration after treatment to be a significant improvement. This can slow or stop the disease progression, preventing further c

Stem Cell Therapy in Duchenne muscular dystrophy (DMD)

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Duchenne muscular dystrophy (DMD) is a genetic disorder characterized by progressive muscle degeneration and weakness due to the alterations of a protein called dystrophin that helps keep muscle cells intact. DMD is one of four conditions known as dystrophinopathies. Duchenne muscular dystrophy (DMD) is a severe type of muscular dystrophy that primarily affects boys. Muscle weakness usually begins around the age of four, and worsens quickly.   Muscle loss typically occurs first in the thighs and pelvis followed by the arms. This can result in trouble standing up. Most are unable to walk by the age of 12. Affected muscles may look larger due to increased fat content. Scoliosis is also common. Some may have intellectual disability.Females with a single copy of the defective gene may show mild symptoms. The disorder is X-linked recessive.   About two thirds of cases are inherited from a person's mother, while one third of cases are due to a new mutation. It is caused by a mutation